dental anatomy : teeth, organs of mastication and defense.
are arranged in two parabolic curves in the upper and lower jaw forming the dental arches. The teeth are located in the alveoli
and fixed by connective tissue. Externally
the tooth is covered by two calcified tissues: cement in root and enamel on the crown. The tooth has three parts: Corona - Neck - Root.
The Crown, covered by enamel is projected into the mouth part is covered by the gums.
Root implanted in the socket is covered with cement. The gum binds tightly to the tooth to the neck.
and fixed by connective tissue. Externally
the tooth is covered by two calcified tissues: cement in root and enamel on the crown. The tooth has three parts: Corona - Neck - Root.
The Crown, covered by enamel is projected into the mouth part is covered by the gums.
Root implanted in the socket is covered with cement. The gum binds tightly to the tooth to the neck.
enamel or enamel substance is the hardest tissue in the body composed mainly of hydroxyapatite crystals and forms the outer layer of the crown.
The cement is formed by cementoblasts. Laminar bone is a substance covering the root.
The cement is formed by cementoblasts. Laminar bone is a substance covering the root.
periodontal ligament collagen fibers formed by fixing the tooth to the socket through.
Dentin forms the internal structure of the tooth. Is amended with fine bone canaliculi arranged radially towards the pulp cavity containing odontoblast.
pulp erupted after a dentin crown all his life to compensate for the wear resulting from mastication.
most common diseases CAUSE PREMATURE WEAR DENTAL
IN THE DOG:
a) enamel hypoplasia
b) CAGE SYNDROME TEETHER
c) formation of plaque and ODONTOLITIASIS PREMATURE: individual predisposition.
d) intrinsic factors: individual immune response
e) Extrinsic factors: habits of life and care of the owner.
enamel loss and subsequent exposure of dentin and / or dental pulp are the gateway to pulp and periapical infections.
Dr. Adalberto Amato. INTEGRAL DENTAL VETERINARY dramato@dentalvet.com.ar --- http://www.dentalvet.com.ar/
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Technical Bulletin 2
Maxillofacial Bone Growth
maxillary bone growth occurs in two forms: displacement by apposition (+) and resorption (-). The surface in the direction of new bone growth receives the opposite is reabsorbed. Growth Areas is formed as a mosaic on the surface of the periosteum (Campos Campos apposition and resorption) generating growth movement (displacement). The bone grows through the soft tissue matrix. The operation of formation and resorption in the growth areas of running the membranes. The bone does not grow by itself. Growth Centers are fields of apposition and resorption of special significance, but the growth occurs in all surfaces. Branch of the mandible grows back as the anterior border remodeling in relation to the body. Mandibular lengthening. This is a genetic factor determining the length of the posterior maxilla and normal occlusion.
Remodeled: As the bone grows moving parts move from one place to another. While some move by resorption and apposition other remodels in relation to this, resulting in elongation growth. Growth brings remodeling.
facial malformations Source:
During the organization of the face, mandibular and maxillary processes emerge from the first gill arch while the frontal and nasal arise from the tissues covering the forebrain. Craniofacial development is dependent on intrinsic genetic and multifactorial polygenic. It is the base of the maxillary growth pathology:
Prognathism and / or braquignatia, both with a polygenic genetic basis.
Other dysfunctions:
Syndrome Pierre - Robin (mandibular hypoplasia) Son defectos de los arcos branquiales (Centros de desarrollo maxilar).
Síndrome de Teacher Collins (Distosis mandíbulo-facial)
Son defectos de los arcos branquiales (Centros de desarrollo maxilar).
Se caracterizan por mandíbula corta (MICROGNATISMO) y curvada, desarrollo incompleto de los arcos malares (Cigoma incompleto)
Se caracterizan por mandíbula corta (MICROGNATISMO) y curvada, desarrollo incompleto de los arcos malares (Cigoma incompleto)
Dentin forms the internal structure of the tooth. Is amended with fine bone canaliculi arranged radially towards the pulp cavity containing odontoblast. pulp erupted after a dentin crown all his life to compensate for the wear resulting from mastication.
most common diseases CAUSE PREMATURE WEAR DENTAL
IN THE DOG:
a) enamel hypoplasia
b) CAGE SYNDROME TEETHER
c) formation of plaque and ODONTOLITIASIS PREMATURE: individual predisposition.
d) intrinsic factors: individual immune response
e) Extrinsic factors: habits of life and care of the owner.
enamel loss and subsequent exposure of dentin and / or dental pulp are the gateway to pulp and periapical infections.
Dr. Adalberto Amato. INTEGRAL DENTAL VETERINARY dramato@dentalvet.com.ar --- http://www.dentalvet.com.ar/
-------------------------------------------------- -------------------------------------------------- ---
Technical Bulletin 2
Maxillofacial Bone Growth
maxillary bone growth occurs in two forms: displacement by apposition (+) and resorption (-). The surface in the direction of new bone growth receives the opposite is reabsorbed. Growth Areas is formed as a mosaic on the surface of the periosteum (Campos Campos apposition and resorption) generating growth movement (displacement). The bone grows through the soft tissue matrix. The operation of formation and resorption in the growth areas of running the membranes. The bone does not grow by itself. Growth Centers are fields of apposition and resorption of special significance, but the growth occurs in all surfaces. Branch of the mandible grows back as the anterior border remodeling in relation to the body. Mandibular lengthening. This is a genetic factor determining the length of the posterior maxilla and normal occlusion.
Remodeled: As the bone grows moving parts move from one place to another. While some move by resorption and apposition other remodels in relation to this, resulting in elongation growth. Growth brings remodeling.
facial malformations Source:
During the organization of the face, mandibular and maxillary processes emerge from the first gill arch while the frontal and nasal arise from the tissues covering the forebrain. Craniofacial development is dependent on intrinsic genetic and multifactorial polygenic. It is the base of the maxillary growth pathology:
Prognathism and / or braquignatia, both with a polygenic genetic basis.
Other dysfunctions:
Syndrome Pierre - Robin (mandibular hypoplasia) Son defectos de los arcos branquiales (Centros de desarrollo maxilar).
Síndrome de Teacher Collins (Distosis mandíbulo-facial)
Son defectos de los arcos branquiales (Centros de desarrollo maxilar).
Se caracterizan por mandíbula corta (MICROGNATISMO) y curvada, desarrollo incompleto de los arcos malares (Cigoma incompleto)
Se caracterizan por mandíbula corta (MICROGNATISMO) y curvada, desarrollo incompleto de los arcos malares (Cigoma incompleto)
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